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BenchBoard

Decision cockpit for pathology foundation-model benchmarking, built to move from broad coverage scans to task-level evidence.

Task Evidence

CPTAC-LUAD · kras_mutation_status (CPTAC-LUAD__KRAS_mutation)

Biomarker & Molecular Profiling / Genomic, Epigenetic & Pathway Alterations

Inspect benchmark evidence for one task at a time: who leads, how stable the split-level results are, and which neighbouring tasks are likely to transfer intuition.

clsL1 · Biomarker & Molecular ProfilingL2 · Genomic, Epigenetic & Pathway AlterationslungH&EInformative evidence

Task metadata

Fields sourced from benchmark_main. Missing values are shown as —.

cls
Identity
task_key
CPTAC-LUAD__KRAS_mutation
dataset_name
CPTAC-LUAD
task_name
kras_mutation_status
task_type
cls
Taxonomy
task_family_l1
Biomarker & Molecular Profiling
task_family_l2
Genomic, Epigenetic & Pathway Alterations
label_structure
binary
num_classes
2
labels
{"0": "negative", "1": "positive"}
Biology
organ
lung
stain
H&E
specimen_type
resection
timepoint
pre_treatment
disease
肺癌
Scale & Labels
patient_n
102
slide_n
312
label_source
molecular_omics
label_visibility
indirect
Result Evidence
result_tier
Informative
result_quality_score
75.2
result_flags
config_sensitive
result_tier_reason
Clear model separation: significant-win density 0.235.Difficulty sits in a usable range: best mean 0.730.Leader changes across checkpoint strategies: 3 distinct leaders.
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Ranked by shared L2 subtype first, then L1 family, organ, type and label structure.